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Explore  /  Noopept (Omberacetam)
Research reference — not for sale

Noopept (Omberacetam)

B
best evidence
Small molecule (non-peptide)
also called — Noopept · GVS-111 · omberacetam · N-phenylacetyl-L-prolylglycine ethyl ester. INCI: none
nootropic / cognitiveneuroprotectionBDNF/NGF (approved in Russia: cognitive disorders)

Reference entry — not sold here. Like Semax (#41) and Selank (#42), an approved nootropic in Russia — and the oral one. No dosing published here.

In brief

Noopept is a dipeptide-derived small molecule (not a classical peptide, despite the ubiquitous label) developed in Russia and approved there as an oral nootropic. It's a prodrug of the endogenous dipeptide cycloprolylglycine, raises BDNF/NGF, and is ~1000× more potent by weight than piracetam. Its strongest evidence is in people with existing cognitive deficits (vascular/traumatic) via Russian trials; the popular healthy-enhancement use and any Western evidence are thin, and it is not FDA-approved. Its practical edge over Semax/Selank is simply that it's oral.

Legal standing, by region
European Union
Not approved

not approved.

United States · your region
Not FDA-approved (gray-market)

Not FDA-approved. Sold as an unregulated "supplement" or research chemical (a legal gray area — the FDA does not recognise it as a lawful dietary ingredient, yet it's widely sold).

International
approval limited to Russia/CIS

approval limited to Russia/CIS.

Evidence, by outcome

An honest grade per outcome — drawn from the evidence, not any catalogue. Hype and undemonstrated marketing claims grade low.

OutcomeEvidence base · effectGrade
Cognitive impairment / MCI (vascular, traumatic — approved Russian use)
Strongest use, but Russian-only, no Western replication; methodology varies from FDA/EMA standards
Russian RCTs (Neznamov & Teleshova 2009, vs piracetam) + EEG studies · Improved memory / cognition
C
BDNF/NGF & neuroprotection mechanism
Animal/mechanistic; human translation is Russian-only
Substantial preclinical (Ostrovskaya, Gudasheva) · Neurotrophic; multi-target
B
Cognitive enhancement in healthy adults (the popular use)
Weaker than the cognitive-deficit use — the inverse of how it's marketed
Extrapolated + anecdotal · Suggestive
D
Anti-amyloid / Alzheimer's
Preclinical
Cellular/animal models (Jia 2011; Ostrovskaya 2014) · Suggestive
D
Anxiolytic
Early
Preclinical (cPG) + some Russian data · Suggestive
D
Safety
Mild (irritability, sleep changes, headache in some); long-term / Western data limited
Approved drug (Russia), oral, broad use · Generally well tolerated

Identity a synthetic dipeptide-derived small molecule built on a Pro-Gly core, designed as a dipeptide analog of the racetam nootropic piracetam. It is a prodrug of cycloprolylglycine (cPG) — an endogenous cyclic dipeptide naturally present in the mammalian brain — which is its main active metabolite. Crucially, it is orally bioavailable and crosses the blood-brain barrier after being swallowed. ## Development & history - Designed in 1996 at the V.V. Zakusov Research Institute of Pharmacology (Russian Academy of Medical Sciences, Moscow) by Tatiana Gudasheva, Sergei Seredenin and colleagues — as a dipeptide analog of piracetam (the hypothesis: piracetam's pyrrolidine ring mimics a proline residue and its N-acetamide mimics glycine).

  • The result was active at ~1000× lower doses than piracetam by weight (~ vs ~) with a broader cognitive + anxiolytic + neuroprotective profile. (Note: the "1000×" reflects potency and mechanism, not 1000× the benefit — a common misreading.)
  • Approved and prescribed in Russia (and neighbouring countries) as a nootropic/neuroprotective medicine. ## Mechanism (as proposed) Noopept is hydrolysed and cyclised to cycloprolylglycine (cPG), an endogenous neuropeptide that modulates AMPA glutamate receptors (key to synaptic transmission and long-term potentiation). It also upregulates BDNF and NGF in the hippocampus (the same neurotrophic theme as Semax/Selank), with cholinergic, antioxidant, anti-inflammatory and HIF-1-mediated (hypoxia-neuroprotective) effects reported. The mechanism is complex, not fully elucidated, and distinct from the classical racetams.
Sources — 4 cited
01Gudasheva TA, et al. (Major brain metabolite of GVS-111 = cyclo-L-prolylglycine; similarity to an endogenous neuropeptide.) Eur J Drug Metab Pharmacokinet. 1997.
02Ostrovskaya RU, et al. Noopept stimulates NGF and BDNF expression in rat hippocampus. Bull Exp Biol Med. 2008; (neuroprotection in an AD cellular model — apoptosis/tau). J Biomed Sci. 2014.
03Neznamov GG, Teleshova ES. Comparative studies of Noopept and piracetam in mild cognitive disorders of vascular and traumatic origin. Neurosci Behav Physiol. 2009.
04Vakhitova YV, et al. (HIF-1 as a primary molecular mechanism of the Pro-Gly dipeptide Noopept.) 2016; Jia X, et al. (Noopept rescues α-synuclein amyloid cytotoxicity.) J Mol Biol. 2011.
Review status
Not yet reviewed

A credentialed reviewer (PharmD / PhD / MD) will be named before this entry is finalised. Until then, treat it as a working draft. Last updated July 2026.

Grades reflect the published evidence, not our interest. No dosing, reconstitution, or administration is published for research compounds — that restraint is deliberate.

Related compounds
SemaxB
Peptide
SelankB
Peptide
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Noopept — evidence, why it's not a peptide & status · Vallydia