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Research reference — not for sale

Amycretin

B
best evidence
Peptide
also called — amycretin
metabolicobesity / weighttype-2 diabetes (investigational)

Reference entry — NOT a sellable lane, NOT approved. The hottest emerging obesity molecule: it folds the CagriSema idea (GLP-1 + amylin) into one molecule, in both oral and injectable forms. Investigational; racing into Phase 3. No dosing published here.

In brief

Amycretin is Novo Nordisk's first-in-class unimolecular GLP-1 + amylin agonist — the CagriSema concept in a single molecule, developed as both a pill and a weekly injection. Early-phase data are striking (~24% weight loss subcutaneously at 36 weeks; ~13% with the oral form at just 12 weeks, beating semaglutide at that timepoint), and Novo is taking both forms straight to Phase 3 in 2026. But it is early-stage (Phase 1b/2a) and not approved anywhere — the impressive numbers still need Phase 3 confirmation.

Legal standing, by region
International
See note

- Everywhere — investigational, not approved. Not available by prescription or compounding outside clinical trials. - Gray market: "research" amycretin sold online is unapproved and unregulated — unknown purity/dose (and, being early-stage, authenticity is especially doubtful). - This venture: not a sellable lane — an investigational drug.

Evidence, by outcome

An honest grade per outcome — drawn from the evidence, not any catalogue. Hype and undemonstrated marketing claims grade low.

OutcomeEvidence base · effectGrade
Weight loss — subcutaneous
Early-phase (Phase 1b/2a), small; needs Phase 3 confirmation; high trial withdrawal
Phase 1b/2a (Lancet 2025, → −24.3% at 36 wk) · Very large
B
Weight loss — oral
Early, short (12 wk); Phase 3 pending
Phase 1 (Lancet 2025, ~13.1% at 12 wk, > semaglutide's ~6%) · Strong for an oral agent
B
Type-2 diabetes (HbA1c)
Phase 2; not approved
Phase 2 (n=448, up to −1.8%) · Meaningful
B
Safety
GI events frequent (as with GLP-1/amylin); long-term profile unestablished; investigational
Phase 1/2 (GI-predominant, mostly mild-moderate) · Class-consistent

Identity a single engineered peptide that activates both the GLP-1 receptor and the amylin receptor — the two pathways that CagriSema (#5) combines using two molecules, here united in one. Developed in both a once-daily oral tablet and a once-weekly subcutaneous injection. ## Mechanism (as proposed) a single molecule agonising both the GLP-1 receptor (hypothalamic appetite control, glucose-dependent insulin, glucagon suppression, slowed gastric emptying) and the amylin receptor (brainstem/area-postrema satiety, gastric emptying, glucagon) — the same complementary two-pathway logic as CagriSema, delivered in one agent. An orally bioavailable version of this is the headline pharmacology.

Sources — 3 cited
01Gasiorek A, et al. Safety, tolerability, PK/PD of the first-in-class GLP-1 and amylin receptor agonist amycretin: a first-in-human Phase 1 study (oral). Lancet. 2025.
02Dahl K, et al. Amycretin, a novel unimolecular GLP-1 and amylin receptor agonist administered subcutaneously: a Phase 1b/2a study. Lancet. 2025.
03Novo Nordisk — Phase 2 T2D topline (2025–2026); Phase 3 advancement announcement (both oral + subcutaneous, Q1 2026).
Review status
Not yet reviewed

A credentialed reviewer (PharmD / PhD / MD) will be named before this entry is finalised. Until then, treat it as a working draft. Last updated July 2026 (fast-moving — re-check Phase 3 progress).

Grades reflect the published evidence, not our interest. No dosing, reconstitution, or administration is published for research compounds — that restraint is deliberate.

Related compounds
CagrilintideB
Peptide
SemaglutideA
Peptide
RetatrutideB
Peptide
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Amycretin — evidence, the "CagriSema in one molecule" story & status · Vallydia